Promyelocytic leukemia nuclear bodies behave as DNA damage sensors whose response to DNA double-strand breaks is regulated by NBS1 and the kinases ATM, Chk2, and ATR

Graham Dellaire, Reagan W. Ching, Kashif Ahmed, Farid Jalali, Kenneth C.K. Tse, Robert G. Bristow, David P. Bazett-Jones

Producción científica: Contribución a una revista › Artículo › revisión exhaustiva

148 Citas (Scopus)

Huella

Profundice en los temas de investigación de 'Promyelocytic leukemia nuclear bodies behave as DNA damage sensors whose response to DNA double-strand breaks is regulated by NBS1 and the kinases ATM, Chk2, and ATR'. En conjunto forman una huella única.

Medicine & Life Sciences