Bone turnover markers in the management of postmenopausal osteoporosis

Jacques P. Brown, Caroline Albert, Bassam A. Nassar, Jonathan D. Adachi, David Cole, K. Shawn Davison, Kent C. Dooley, Andrew Don-Wauchope, Pierre Douville, David A. Hanley, Sophie A. Jamal, Robert Josse, Stephanie Kaiser, John Krahn, Richard Krause, Richard Kremer, Raymond Lepage, Elaine Letendre, Suzanne Morin, Daylily S. OoiAlexandra Papaioaonnou, Louis Georges Ste-Marie

Résultat de recherche: Review articleexamen par les pairs

180 Citations (Scopus)

Résumé

Osteoporosis is the most common cause of fragility fractures. Bone remodelling is essential for repairing damaged areas within bone to preserve bone strength and for assisting in mineral homeostases. In young adults, bone remodelling is usually balanced with approximately as much bone replaced as is removed during each remodelling cycle. However, when remodelling becomes accelerated in combination with an imbalance that favours bone resorption over formation, such as during menopause, precipitous losses in bone mass occur. Bone turnover markers (BTMs) measure the rate of bone remodelling allowing for a dynamic assessment of skeletal status and hold promise in identifying those at highest risk of rapid bone loss and subsequent fracture. Further, the use of BTMs to monitor individuals administered osteoporosis therapy is attractive as monitoring anti-fracture efficacy with bone mineral density has significant limitations. This review details remodelling biology, pre-analytical and analytical sources of variability for BTMs, describes the most commonly used resorption and formation markers, and offers some guidelines for their use and interpretation in the laboratory and the clinic.

Langue d'origineEnglish
Pages (de-à)929-942
Nombre de pages14
JournalClinical Biochemistry
Volume42
Numéro de publication10-11
DOI
Statut de publicationPublished - juill. 2009

Note bibliographique

Funding Information:
This project was a result of an unrestricted grant from Osteoporosis Canada. The funding source had no role in the development of these recommendations.

ASJC Scopus Subject Areas

  • Clinical Biochemistry

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