Emphysema is-at the most-only a mild phenotype in the sugen/hypoxia rat model of pulmonary arterial hypertension

Harm J. Bogaard; Ekaterina Legchenko; Ketul R. Chaudhary; Xiao Qing Sun; Duncan J. Stewart; Georg Hansmann

doi:10.1164/rccm.201906-1200LE

Emphysema is-at the most-only a mild phenotype in the sugen/hypoxia rat model of pulmonary arterial hypertension

Harm J. Bogaard, Ekaterina Legchenko, Ketul R. Chaudhary, Xiao Qing Sun, Duncan J. Stewart, Georg Hansmann

Résultat de recherche: Letter › examen par les pairs

Langue d'origine	English
Pages (de-à)	1447-1450
Nombre de pages	4
Journal	American Journal of Respiratory and Critical Care Medicine
Volume	200
Numéro de publication	11
DOI	https://doi.org/10.1164/rccm.201906-1200LE
Statut de publication	Published - déc. 1 2019
Publié à l'externe	Oui

Note bibliographique

Funding Information:
This article is open access and distributed under the terms of the Creative Commons Attribution Non-Commercial No Derivatives License 4.0 (http:// creativecommons.org/licenses/by-nc-nd/4.0/). For commercial usage and reprints, please contact Diane Gern (dgern@thoracic.org). Supported by an ERS/EU RESPIRE 3 Marie Sklodowska-Curie Postdoctoral Fellowship (E.L.); the Netherlands CardioVascular Research Initiative: the Dutch Heart Foundation, Dutch Federation of University Medical Centers, the Netherlands Organization for Health Research and Development, and the Royal Netherlands Academy of Sciences Grant 2012-08 awarded to the Phaedra consortium (H.J.B.); a Foundation grant from the Canadian Institutes of Health Research (D.J.S.); and the German Research Foundation (DFG; HA4348/2-2 and HA4348/6 KFO311) and the European Pediatric Pulmonary Vascular Disease Network (www.pvdnetwork.org) (G.H.).

Funding Information:
Supported by the Deutsche Forschungsgemeinschaft, Projektnummer 268555672, SFB 1213, Project CP02, A07, and A08.

ASJC Scopus Subject Areas

Pulmonary and Respiratory Medicine
Critical Care and Intensive Care Medicine

PubMed: MeSH publication types

Letter
Research Support, Non-U.S. Gov't
Comment

Accès au document

10.1164/rccm.201906-1200LE

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title = "Emphysema is-at the most-only a mild phenotype in the sugen/hypoxia rat model of pulmonary arterial hypertension",

author = "Bogaard, {Harm J.} and Ekaterina Legchenko and Chaudhary, {Ketul R.} and Sun, {Xiao Qing} and Stewart, {Duncan J.} and Georg Hansmann",

note = "Funding Information: This article is open access and distributed under the terms of the Creative Commons Attribution Non-Commercial No Derivatives License 4.0 (http:// creativecommons.org/licenses/by-nc-nd/4.0/). For commercial usage and reprints, please contact Diane Gern (dgern@thoracic.org). Supported by an ERS/EU RESPIRE 3 Marie Sklodowska-Curie Postdoctoral Fellowship (E.L.); the Netherlands CardioVascular Research Initiative: the Dutch Heart Foundation, Dutch Federation of University Medical Centers, the Netherlands Organization for Health Research and Development, and the Royal Netherlands Academy of Sciences Grant 2012-08 awarded to the Phaedra consortium (H.J.B.); a Foundation grant from the Canadian Institutes of Health Research (D.J.S.); and the German Research Foundation (DFG; HA4348/2-2 and HA4348/6 KFO311) and the European Pediatric Pulmonary Vascular Disease Network (www.pvdnetwork.org) (G.H.). Funding Information: Supported by the Deutsche Forschungsgemeinschaft, Projektnummer 268555672, SFB 1213, Project CP02, A07, and A08.",

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T1 - Emphysema is-at the most-only a mild phenotype in the sugen/hypoxia rat model of pulmonary arterial hypertension

AU - Bogaard, Harm J.

AU - Legchenko, Ekaterina

AU - Chaudhary, Ketul R.

AU - Sun, Xiao Qing

AU - Stewart, Duncan J.

AU - Hansmann, Georg

N1 - Funding Information: This article is open access and distributed under the terms of the Creative Commons Attribution Non-Commercial No Derivatives License 4.0 (http:// creativecommons.org/licenses/by-nc-nd/4.0/). For commercial usage and reprints, please contact Diane Gern (dgern@thoracic.org). Supported by an ERS/EU RESPIRE 3 Marie Sklodowska-Curie Postdoctoral Fellowship (E.L.); the Netherlands CardioVascular Research Initiative: the Dutch Heart Foundation, Dutch Federation of University Medical Centers, the Netherlands Organization for Health Research and Development, and the Royal Netherlands Academy of Sciences Grant 2012-08 awarded to the Phaedra consortium (H.J.B.); a Foundation grant from the Canadian Institutes of Health Research (D.J.S.); and the German Research Foundation (DFG; HA4348/2-2 and HA4348/6 KFO311) and the European Pediatric Pulmonary Vascular Disease Network (www.pvdnetwork.org) (G.H.). Funding Information: Supported by the Deutsche Forschungsgemeinschaft, Projektnummer 268555672, SFB 1213, Project CP02, A07, and A08.

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