Oxidative energy deficiency. II. Human achondroplasia

B. Mackler; B. Haynes; A. R. Inamdar; L. R. Pedegana; J. G. Hall; M. M. Cohen

doi:10.1016/0003-9861(73)90531-6

Oxidative energy deficiency. II. Human achondroplasia

B. Mackler, B. Haynes, A. R. Inamdar, L. R. Pedegana, J. G. Hall, M. M. Cohen

Résultat de recherche: Article › examen par les pairs

19 Citations (Scopus)

Résumé

Defective oxidative energy formation (with decreased phosphorylation possibly occurring at Site I of phosphorylation, the NADH dehydrogenase region of the terminal respiratory system) has been demonstrated in mitochondrial preparations isolated from muscle biopsy specimens from human subjects with classical achondroplasia.

Langue d'origine	English
Pages (de-à)	885-888
Nombre de pages	4
Journal	Archives of Biochemistry and Biophysics
Volume	159
Numéro de publication	2
DOI	https://doi.org/10.1016/0003-9861(73)90531-6
Statut de publication	Published - déc. 1973
Publié à l'externe	Oui

Note bibliographique

Funding Information:
’ This work was supported in part by Grants HL 03437, Hl) 06346, HI) 0.5961, and a career development award (,M.M.C.) from the National Institutes of Healt,h, and Grant CRBS-27.5 from the National Foundation. A portion of this work was conducted through the Clinical Research Center facility of the University of Washington, srlpported by the NIH Grant RR-X.

ASJC Scopus Subject Areas

Biophysics
Biochemistry
Molecular Biology

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10.1016/0003-9861(73)90531-6

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title = "Oxidative energy deficiency. II. Human achondroplasia",

abstract = "Defective oxidative energy formation (with decreased phosphorylation possibly occurring at Site I of phosphorylation, the NADH dehydrogenase region of the terminal respiratory system) has been demonstrated in mitochondrial preparations isolated from muscle biopsy specimens from human subjects with classical achondroplasia.",

author = "B. Mackler and B. Haynes and Inamdar, {A. R.} and Pedegana, {L. R.} and Hall, {J. G.} and Cohen, {M. M.}",

note = "Funding Information: {\textquoteright} This work was supported in part by Grants HL 03437, Hl) 06346, HI) 0.5961, and a career development award (,M.M.C.) from the National Institutes of Healt,h, and Grant CRBS-27.5 from the National Foundation. A portion of this work was conducted through the Clinical Research Center facility of the University of Washington, srlpported by the NIH Grant RR-X.",

year = "1973",

month = dec,

doi = "10.1016/0003-9861(73)90531-6",

language = "English",

volume = "159",

pages = "885--888",

journal = "Archives of Biochemistry and Biophysics",

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T1 - Oxidative energy deficiency. II. Human achondroplasia

AU - Mackler, B.

AU - Haynes, B.

AU - Inamdar, A. R.

AU - Pedegana, L. R.

AU - Hall, J. G.

AU - Cohen, M. M.

N1 - Funding Information: ’ This work was supported in part by Grants HL 03437, Hl) 06346, HI) 0.5961, and a career development award (,M.M.C.) from the National Institutes of Healt,h, and Grant CRBS-27.5 from the National Foundation. A portion of this work was conducted through the Clinical Research Center facility of the University of Washington, srlpported by the NIH Grant RR-X.

PY - 1973/12

Y1 - 1973/12

N2 - Defective oxidative energy formation (with decreased phosphorylation possibly occurring at Site I of phosphorylation, the NADH dehydrogenase region of the terminal respiratory system) has been demonstrated in mitochondrial preparations isolated from muscle biopsy specimens from human subjects with classical achondroplasia.

AB - Defective oxidative energy formation (with decreased phosphorylation possibly occurring at Site I of phosphorylation, the NADH dehydrogenase region of the terminal respiratory system) has been demonstrated in mitochondrial preparations isolated from muscle biopsy specimens from human subjects with classical achondroplasia.

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JO - Archives of Biochemistry and Biophysics

JF - Archives of Biochemistry and Biophysics

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ER -

Oxidative energy deficiency. II. Human achondroplasia

Résumé

Note bibliographique

ASJC Scopus Subject Areas

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