Résumé
Streptococcus mutans NG5 failed to anchor antigen P1 to the cell surface, and such a failure could be attributed to a defective SrtA, which was made defective by a point mutation within the srtA gene. Without a functional SrtA, S. mutans NG5 was not able to perform a number of cell surface-related activities, including saliva-mediated adherence and aggregation.
Langue d'origine | English |
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Pages (de-à) | 4314-4317 |
Nombre de pages | 4 |
Journal | Infection and Immunity |
Volume | 72 |
Numéro de publication | 7 |
DOI | |
Statut de publication | Published - juill. 2004 |
ASJC Scopus Subject Areas
- Parasitology
- Microbiology
- Immunology
- Infectious Diseases
PubMed: MeSH publication types
- Journal Article
- Research Support, Non-U.S. Gov't
Empreinte numérique
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Lee, S. F., & McGavin, M. K. H. (2004). Identification of a point mutation resulting in loss of cell wall anchoring activity of SrtA of Streptococcus mutans NG5. Infection and Immunity, 72(7), 4314-4317. https://doi.org/10.1128/IAI.72.7.4314-4317.2004